Table 2

Established (E) and possible (P) sequelae of foodborne infections that will be assessed in this study (British Columbia, Canada)

Foodborne infection*	Acute kidney injury	Coeliac disease	Erythema nodosum	Graves’ disease	Guillain-Barré syndrome†	Haemolytic uremic syndrome	Inflammatory bowel disease	Irritable bowel syndrome	Neonatal listeriosis‡	Stillbirth	Reactive arthritis§	Thrombotic thrombocytopenic purpura¶
Campylobacter	E	P			E	P	P	P			E
Cryptosporidium											P
Cyclospora					P						P
Giardia								P			P
Hepatitis A virus	E				P
Listeria monocytogenes									E ¶	E
Salmonella (non-typhoidal)	E				P	P	P	P			E
Salmonella Paratyphi	E				P	P	P	P			E
Salmonella Typhi	E				P	P	P	P			E
STEC	E					E					P	P
Shigella	E					E		P			E	P
Yersinia (excluding pestis)	E		E	P				P			E

*Clostridium botulinum and Vibrio parahaemolyticus do not have established or possible sequelae.
†This includes GBS variants such as Miller Fisher syndrome; other neurological conditions such as chronic inflammatory demyelinating polyneuropathy will also be assessed.
‡Considered here as a sequela of maternal Listeria infection.
§This includes associated diagnoses such as anterior uveitis and ankylosing spondylitis.
¶Shown not to be a sequela; retained to capture historical misdiagnosis of HUS.
GBS, Guillain-Barré syndrome; HUS, haemolytic uremic syndrome; STEC, Shiga toxin-producing Escherichia coli.