The proinflammatory heart failure phenotype: a case of integrative physiology

Am J Med Sci. 2005 Nov;330(5):219-26. doi: 10.1097/00000441-200511000-00004.
No abstract available

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Aldosterone / blood
  • Animals
  • Calcium / physiology
  • Coronary Circulation / physiology*
  • Cytokines / immunology
  • Disease Models, Animal
  • Heart Failure / blood
  • Heart Failure / etiology*
  • Heart Failure / immunology
  • Heart Failure / physiopathology
  • Humans
  • Hyperaldosteronism / blood
  • Hyperaldosteronism / complications
  • Hyperaldosteronism / immunology
  • Hyperparathyroidism / blood
  • Hyperparathyroidism / complications
  • Hyperparathyroidism / immunology
  • Inflammation / complications
  • Inflammation / immunology*
  • Magnesium / physiology
  • Oxidative Stress / immunology
  • Phenotype

Substances

  • Cytokines
  • Aldosterone
  • Magnesium
  • Calcium